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ATP5O — EGF
Text-mined interactions from Literome
Falin et al., Am J Physiol Cell Physiol 2005
:
The DBHQ mediated decrease in I ( sc ) was due to inhibition of basolateral Na ( + ) -K ( + )
-ATPase , but
EGF- , ATP-, and PMA induced inhibition was most likely
due to reduced apical sodium entry ( epithelial Na ( + ) channel activity )
Nitsche et al., Am J Physiol Gastrointest Liver Physiol 2007
:
BMP-4 attenuated
EGF mediated inhibition of H ( + ) /K ( + )
-ATPase gene expression and blocked EGF induction of both parietal cell morphological transformation and MAPK activation
Hughes et al., Mol Pharmacol 1991
:
The Ca ( 2+ )
-ATPase inhibitor thapsigargin also activated Ca2+ entry, and neither
epidermal growth factor nor the guanine nucleotide dependent protein linked receptor agonist bradykinin
activated additional Ca2+ entry over that due to thapsigargin
Knowles et al., Arch Biochem Biophys 1990
(Carcinoma, Hepatocellular...) :
The tyrosine kinase activity of the
EGF receptor was
essential for the induction of ectoCa2 ( + )
-ATPase , since enzyme induction was abolished by a tyrosine kinase inhibitor, genistein
Knowles et al., Biochem Biophys Res Commun 1985
(Carcinoma, Hepatocellular...) :
The increased expression of the
ectoCa2+-ATPase was absolutely
dependent on
EGF , but also required hydrocortisone and cholera toxin
Kaise et al., J Biol Chem 1995
:
EGF in concentrations equivalent to those in plasma
increased H+, K ( + )
-ATPase alpha-subunit mRNA levels ... Our studies indicate that
EGF induces gastric H+, K ( + )
-ATPase alpha-subunit gene expression via an interaction between a specific ERE and a novel transcriptional factor and that this may be a physiologic mechanism by which EGF regulates acid secretion
Soler et al., Regul Pept 1994
:
Effects of
epidermal growth factor on gluconeogenesis and cellular redox state do not
require Na+/H+ exchange or Na+/K ( + )
-ATPase activities ... Although Na+/H+ exchange and/or Na+/K ( + )
-ATPase are
regulated by
EGF , our results indicate that these activities are not required for the effects of EGF on gluconeogenesis and/or cytosolic and mitochondrial redox state
Borok et al., Am J Physiol 1996
:
Net sodium flux and Na+, K+
-ATPase activity both
increased approximately 50 % in the presence of
EGF
Mehta et al., JPEN J Parenter Enteral Nutr 1997
:
Effects of
EGF on the kinetic parameters of sodium-glucose contransporter, thymidine transport, and on the activity of Na+/K ( + )
-ATPase were examined