Gene interactions and pathways from curated databases and text-mining

◀ Back to IL10

IKBKB — IL10

Text-mined interactions from Literome

Li et al., J Exp Med 1999 : Accordingly, IKKbeta-deficient cells are defective in activation of IKK and NF-kappaB in response to either tumor necrosis factor alpha or interleukin 1
Bhattacharyya et al., Blood 2004 : Furthermore, IL-10 blocked inducible Akt phosphorylation, and inhibitors of phosphatidylinositol 3-kinase (PI3K) effectively suppressed the activation of Akt, IKK , and NF-kappaB
Lamothe et al., J Biol Chem 2007 : Reconstituting TRAF6-deficent cells with various TRAF6 mutants, we clearly demonstrate the requirement for the TRAF6 RING domain and site-specific auto-ubiquitination of TRAF6 to activate IKK in response to interleukin-1
Ropelle et al., PLoS Biol 2010 (Hyperphagia...) : This molecular mechanism, mediated by physical activity, involves the anti-inflammatory protein IL-10 , a core inhibitor of IKKbeta/NF-kappaB signaling and ER stress
Sarkar et al., Mol Cell Biol 2011 : RNA binding protein AUF1 regulates lipopolysaccharide induced IL10 expression by activating IkappaB kinase complex in monocytes
O'Connell et al., J Biol Chem 1998 : Recently, we have reported that the tumor necrosis factor and interleukin 1 signaling pathways activate two kinases, IKK1 and IKK2 ... Recently, we have reported that the tumor necrosis factor and interleukin 1 signaling pathways activate two kinases, IKK1 and IKK2