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ANGPT2 — BAX

Text-mined interactions from Literome

Bhaskaran et al., American journal of physiology. Renal physiology 2003 : In addition, ANG II increased RPTEC expression of Bax , a cell death protein
Lee et al., J Cell Sci 2010 (Pulmonary Fibrosis) : Ang II caused cytochrome c release from the mitochondria concurrent with caspase-3 activation and DNA fragmentation, and apoptosis was suppressed by an inhibitor of Bax-protein channel formation, implicating mitochondrial mediated apoptosis
Ren et al., Journal of biomedical research 2012 : Specifically, angiopoietin-1 prevented DOX induced increases in FasL and Bax levels and cleaved caspase-3 and caspase-8 levels in H9C2 cells