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CSF3 — NFKB1

Text-mined interactions from Literome

Nishiki et al., Am J Physiol Cell Physiol 2004 (MAP Kinase Signaling System) : G-CSF did not affect LPS induced activation of ERK, p38, JNK, and NF-kappa B , indicating that G-CSF affects the pathway downstream or independently of these signaling molecules
Xiao et al., Mol Pharmacol 2007 : The effects of 5-AED on survival and G-CSF secretion were blocked by the NFkappaB inhibitor N-benzoyloxycarbonyl ( Z ) -Leu-Leu-leucinal ( MG132 )
Scicchitano et al., Blood Cells Mol Dis 2008 (MAP Kinase Signaling System) : In addition, these results suggest that IL-6, IL-11, GM-CSF, G-CSF and Activin A are similarly regulated by p38 and NF-kappaB and that the MEK1, JNK and PKC pathways appear to play a more limited role in modulating cytokine expression in HBMSC
Segal et al., PloS one 2010 (Granulomatous Disease, Chronic...) : When challenged with either intratracheal zymosan or LPS, NADPH oxidase-deficient p47 ( phox-/- ) mice and gp91(phox)-deficient mice developed exaggerated and progressive lung inflammation, augmented NF-kappaB activation , and elevated downstream pro-inflammatory cytokines ( TNF-alpha, IL-17, and G-CSF ) compared to wildtype mice
Himes et al., Oncogene 1993 : HTLV-1 tax activation of the GM-CSF and G-CSF promoters requires the interaction of NF-kB with other transcription factor families