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MUC17 — PPARG
Text-mined interactions from Literome
Slomiany et al., IUBMB Life 2003
(Helicobacter Infections) :
Using gastric mucosal cells in culture, we show that activation of
PPARgamma with a specific synthetic agonist, ciglitazone,
prevents in a dose dependent fashion ( up to 90.2 % ) the LPS induced reduction in
mucin synthesis, and the effect is reflected in a marked decrease in the LPS induced apoptosis ( 72.4 % ), NO generation ( 80.1 % ), and the expression of NOS-2 activity ( 90 % )
Slomiany et al., IUBMB Life 2004
:
We show that activation of
PPARgamma with a specific agonist, ciglitazone,
prevents the LPS induced reduction in
mucin synthesis, and the effect is reflected in a marked decrease in apoptosis, caspase-3 activity and NO generation
Slomiany et al., Inflammopharmacology 2004
:
Using gastric mucosal cells in culture, we show that activation of
PPARgamma with a specific agonist, ciglitazone,
prevents the LPS induced reduction in
mucin synthesis, and the effect is reflected in a marked decrease in apoptosis, caspase-3 activity and NO generation
Lee et al., Am J Physiol Lung Cell Mol Physiol 2006
(Carcinoma, Mucoepidermoid...) :
However, no data are available on the
role of
PPAR-gamma in smoke induced
mucin production ... In this study, we investigated the
effect of
PPAR-gamma agonist ( rosiglitazone ) on smoke induced
mucin production in NCI-H292 cells