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MAPK3 — S100A9

Pathways - manually collected, often from reviews:

• OpenBEL Selventa BEL large corpus: MAPK3 → S100A9 (increases, S100A9 Activity)
  Evidence: S100-A9 binds the transcription factors NF-kB and P53, and activates the P38 and ERK MAPK kinases and the receptor for advanced glycated end products (RAGE) (50).

Text-mined interactions from Literome

Simard et al., J Leukoc Biol 2010 : S100A9 induced the phosphorylation of the MAPKs, ERK1/2 , p38, and JNK
Schenten et al., Biochim Biophys Acta 2010 : Inhibition of p38 MAPK activity and S100A9 phosphorylation by bromoenol lactone, a selective inhibitor of iPLA ( 2 ), indicated that p38 MAPK mediated S100A9 phosphorylation is dependent on iPLA ( 2 )
Bando et al., Biochim Biophys Acta 2013 : Modeled using human epidermal keratinocytes ( HaCaT cells ), IL-1a stimulated the phosphorylation of p38 MAPK and induced S100A9 expression, which was blocked by IL-1 receptor antagonist, RNAi suppression of p38, or a p38 MAPK inhibitor