◀ Back to BDNF

BDNF — CAMK4

Text-mined interactions from Literome

Takeuchi et al., J Neurochem 2002 (Neuroblastoma) : The exon IV-BDNF promoter activity was increased by transfection with CaM KII, MEKK and PKA, but not by CaM KIV
Suzuki et al., J Neurosci 2005 : Consequently, when calcineurin was inhibited by its inhibitor FK506 under the depolarizing condition, the CaMK mediated increase in BDNF became a stimulatory signal, and the endogenous BDNF autocrine system was capable of upregulating NR2C mRNA via the common TrkB-ERK cascade
Spencer et al., Mol Cell Neurosci 2008 : BDNF activates CaMKIV and PKA in parallel to block MAG mediated inhibition of neurite outgrowth
Blanquet et al., J Biol Chem 1997 : In addition, we find that : ( i ) the time course of BDNF action is not accompanied by a change in the spectrum of either alpha- and beta-subunits of CaMK2 detected by immunoblotting ; ( ii ) both treatment of solubilized CaMK2 with alkaline phosphatase and treatment of immunoprecipitated CaMK2 with protein phosphatase 1 reverse phosphorylation and activation of the kinase ; ( iii ) phospholipase C inhibitor D609 and intracellular Ca2+ chelation by 1,2-bis- ( o-aminophenoxy ) ethane-N, N,N '', N ', -tetracetic acid tetra ( acetoxymethyl ) ester or 8- ( diethylamino ) octyl-3,4,5-trimethoxybenzoate but not omission of Ca2+ or Ca2+ chelation by EGTA, abolish the stimulatory effect of BDNF on phosphorylation and activation of CaMK2