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DMD — NOS3

Text-mined interactions from Literome

Bia et al., J Mol Cell Cardiol 1999 (Body Weight...) : Endothelial NOS (eNOS) activity was not affected by dystrophin loss, but inducible NOS (iNOS) activity was seven-fold higher than control in the mdx mouse heart by 12 months of age
Gücüyener et al., Brain Dev 2000 (Cerebral Palsy...) : Nitric oxide is formed in skeletal muscle by the neuronal type nitric oxide synthase and the signalling function of dystrophin and related compounds are in part mediated by nitric oxide
Sander et al., Proc Natl Acad Sci U S A 2000 (Ischemia...) : In both mouse and human skeletal muscle, dystrophin deficiency results in loss of neuronal nitric oxide synthase , which normally is localized to the sarcolemma as part of the dystrophin-glycoprotein complex
Thomas et al., PloS one 2012 (Disease Models, Animal...) : In patients with Duchenne muscular dystrophy (DMD) and the standard mdx mouse model of DMD, dystrophin deficiency causes loss of neuronal nitric oxide synthase ( nNOSµ ) from the sarcolemma, producing functional ischemia when the muscles are exercised