Sakurai et al., FEBS Lett 2005
:
The RE/AP-driven transcription was regulated by TAK1 mediated activation of the c-Jun NH2-terminal kinase, p38 and IkappaB kinase
Shin et al., Biochim Biophys Acta 2009
:
The phosphorylation of TAB1 was occurred at Ser-423 and Thr-431, the residues underlying the p38 mediated feedback inhibition of TAK1 Tran et al., J Biol Chem 2012
(MAP Kinase Signaling System) :
In agreement with this, overexpression of ASK1 or TAK1 resulted in enhanced p38MAPK activation, and their knockdown inhibited p38MAPK in C2C12 cells