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PI3 — PLCG2

Text-mined interactions from Literome

Bobe et al., Blood 2001 (Agammaglobulinemia) : In conclusion, these results demonstrate that in mouse megakaryocytes, CRP leads to PI 3-kinase dependent translocation of PLCgamma2 and Btk that are independent of one another, whereas thrombin only induces translocation of Btk through a pathway that is independent of PI 3-kinase activity
Yamamori et al., Biochem Biophys Res Commun 2004 : Furthermore, PI3K inhibitors reduced the activation of phospholipase Cgamma2 without affecting tyrosine phosphorylation on it
Dearden-Badet et al., Cell Signal 2005 : Both events were dependent on cytoskeleton integrity and Src kinase activity, but only PLC-gamma2 phosphorylation did not require PI3-kinase activity
Mueller et al., Blood 2010 : We report here that the signaling pathway downstream of Syk divides into a phospholipase C (PLC) gamma2- and phosphoinositide 3-kinase (PI3K) gamma dependent pathway
Gratacap et al., J Biol Chem 1998 : We propose that PI 3-kinase and its product PtdIns ( 3,4,5 ) P3 play a key role in the activation and adequate location of PLCgamma2 induced by FcgammaRIIA cross linking