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NTRK1 — PI3

Text-mined interactions from Literome

Bilderback et al., J Neurochem 2001 : The phosphoinositide 3-kinase ( PI 3-kinase ) inhibitor, LY294002, reversed this inhibition suggesting that Trk A activation of PI 3-kinase is necessary to inhibit sphingolipid signaling by p75 ( NTR )
Oda et al., Infect Immun 2006 (MAP Kinase Signaling System) : K252a, an inhibitor of TrkA receptor, and LY294002, an inhibitor of phosphatidylinositol 3-kinase (PI3K) , reduced the toxin induced production of O2 ( - ) and phosphorylation of PDK1, but not the formation of DG
Cattaneo et al., International journal of molecular sciences 2013 : Upon activation, intracellular domains of FPR2 mediate signaling to G-proteins, which trigger several agonist dependent signal transduction pathways, including activation of phospholipase C (PLC), protein kinase C ( PKC ) isoforms, the phosphoinositide 3-kinase (PI3K)/protein kinase B ( Akt ) pathway, the mitogen activated protein kinase ( MAPK ) pathway, p38MAPK, as well as the phosphorylation of cytosolic tyrosine kinases, tyrosine kinase receptor transactivation , phosphorylation and nuclear translocation of regulatory transcriptional factors, release of calcium and production of oxidants