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JUN — VCAM1

Text-mined interactions from Literome

Cui et al., Zhonghua Jie He He Hu Xi Za Zhi 2002 (Asthma...) : NF-kappa B and AP-1 may play important roles in the regulation of VCAM-1 and eotaxin
Tsou et al., Toxicol Appl Pharmacol 2005 : On the other hand, the TNF-alpha induced VCAM-1 expression could be completely abolished by inhibition of AP-1 or NF-kappaB activity, suggesting that activation of both AP-1 and NF-kappaB was necessary for VCAM-1 expression
Hosokawa et al., Clin Exp Immunol 2006 : Interestingly, VCAM-1 expression was enhanced by MEK inhibitor ( PD98059 ) and c-Jun NH2-terminal kinase (JNK) inhibitor ( SP600125 )
Hosokawa et al., Clin Exp Immunol 2006 (Periodontitis) : However, mitogen activated protein kinase ( MEK ) and c-Jun NH2-terminal kinase (JNK) inhibitor enhanced only VCAM-1 expression on HGF
Lin et al., PloS one 2012 (Osteoarthritis) : CCL2 mediated VCAM-1 expression was attenuated by CCR2 inhibitor ( RS102895 ), PKCd inhibitor ( rottlerin ), p38MAPK inhibitor ( SB203580 ), and AP-1 inhibitors ( curcumin and tanshinone IIA )
Liu et al., Biochim Biophys Acta 2013 (Osteoarthritis) : CCN4 induced VCAM-1 expression was attenuated by avß5 or a6ß1 integrin antibody, Syk inhibitor, PKCd inhibitor ( rottlerin ), JNK inhibitor ( SP600125 ), and AP-1 inhibitors ( curcumin and tanshinone )
Udagawa et al., J Immunol 1996 : Similar to expression of IL-2, we demonstrated that recombinant vascular-cell adhesion molecule-1 , when co-immobilized with anti-CD3 mAb, significantly enhanced the induction of transcription factors NF-AT, AP-1 , and NF-kappa B as determined by electromobility shift assays
Ahmad et al., J Biol Chem 1998 : Since c-Fos/c-Jun ( activating protein-1 (AP-1) ) are expressed in vascular endothelium during proinflammatory conditions, we investigated the role of AP-1 proteins in the expression of VCAM-1 by TNF-alpha in SV40 immortalized human microvascular endothelial cells ( HMEC )