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ATP5O — IL1A
Text-mined interactions from Literome
Tattersall et al., Cell Physiol Biochem 2005
:
The
upregulation of
ATPase activity by
IL-1 was inhibited by the NOS inhibitor L-NAME and by the NO scavenger PTIO
Chan et al., J Food Sci 2009
(Neurodegenerative Diseases) :
H ( 2 ) O ( 2 ) or MPP ( + ) treatment significantly decreased Na ( + ) -K ( + ) -ATPase activity, elevated caspase-3 activity and levels of
interleukin (IL)-1 , IL-6, and tumor necrosis factor (TNF)-alpha ( P < 0.05 ) ; and the pretreatments from these agents significantly
restored Na ( + ) -K ( + )
-ATPase activity, suppressed caspase-3 activity and release of IL-1, IL-6, and TNF-alpha ( P < 0.05 )
Bulut et al., Ulus Travma Acil Cerrahi Derg 2011
(Pancreatitis) :
While plasma amylase, lipase,
IL-1ß , and TNF-a levels, and tissue MDA and MPO levels significantly increased in rats with cerulean induced AP, tissue GSH and
Na+-K+-ATPase activity significantly
reduced
Brisseau et al., J Biol Chem 1996
:
Finally, stimulation of both cAMP dependent protein kinase and protein kinase C was required for the stimulatory
effect of
IL-1 on
V-ATPase activity
McTiernan et al., Circ Res 1997
:
The effects on transcript levels were gene specific
; IL-1 beta induced transcripts for inducible NO synthase (iNOS), did not alter GAPDH transcripts, and
reduced sarcoplasmic reticulum Ca ( 2+ )
-ATPase ( 65 % of control, P < .001 ) transcripts