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MRXS5 — TNF

Text-mined interactions from Literome

la Sala et al., J Immunol 2001 (Inflammation) : ATP inhibition of TNF-alpha and IL-12 production by mature DCs was not mediated by PGs or elevation of intracellular cAMP and did not require ATP degradation
Kniss et al., Placenta 2001 : These cells exhibit many of the properties of villous or extravillous trophoblasts and produce large amounts of PGs in response to TNF-alpha
Matsuwaki et al., J Endocrinol 2004 : The inhibitory effect of TNF-alpha on the LH surge was blocked by pretreatment with indomethacin, suggesting that the effects of TNF-alpha were mediated by prostaglandins (PGs)
Rouzer et al., J Biol Chem 2004 : These results demonstrate autocrine regulation of TNF-alpha secretion by endogenous PGs synthesized primarily by COX-1 in RPM and suggest that COX-1 may play a significant role in the regulation of the early response to endotoxemia
Woclawek-Potocka et al., J Reprod Dev 2004 : When the cells were exposed to TNFalpha in combination with NOS inhibitor ( L-NAME ), TNFalpha stimulated PGs production was reduced ( P < 0.05 )
Blitek et al., Reprod Domest Anim 2006 : Summarizing, TNFalpha induces both PGs secretion from cultured porcine endometrium, but preferentially stimulates PGF ( 2alpha ) release from luminal epithelial cells
Romero et al., Prostaglandins Leukot Essent Fatty Acids 1989 : A significant increase in the release of all PGs into the media occurred in response to LPS and TNF
Baud et al., FEBS Lett 1988 : Indomethacin suppressed the effect of TNF on PGs but only reduced that on cAMP, indicating that PG production partly mediates the increase in cAMP