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Pacurari et al., Environ Health Perspect 2008
(Neoplasms, Mesothelial) :
In the present study, we exposed mesothelial cells to SWCNTs and investigated reactive oxygen species ( ROS ) generation, cell viability, DNA damage, histone H2AX phosphorylation, activation of poly(ADP-ribose) polymerase 1 ( PARP-1 ), stimulation of extracellular signal regulated kinase ( ERKs ),
Jun N-terminal kinases (JNKs) , protein p38, and
activation of activator protein-1 (AP-1), nuclear factor kappaB (NF-kappaB), and protein
serine-threonine kinase ( Akt )
Schinkmann et al., J Biol Chem 1997
:
Overexpression of
mst-3 wild-type or kinase dead protein
affects neither the extracellular signal regulated kinases ( ERK1/2 or ERK6 ),
c-Jun N-terminal kinase (JNK) , p38, nor pp70S6 kinase, suggesting that mst-3 is part of a novel signaling pathway