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CSF2 — FCGR1A

Text-mined interactions from Literome

Grattage et al., Immunol Cell Biol 1992 : The major findings were : PMA increased cell surface FcRI, FcRII and CD11b, but decreased HLA ; PMA caused a fivefold increase in all three FcRII RNA transcripts ( 2.5, 1.5 and 0.9 kb ) in Northern analysis ; IFN gamma, IFN alpha and GM-CSF increased the expression of FcRI and II, and there was no effect with IL-1 beta, IL-2, TNF alpha or TNF beta ( only GM-CSF increased the expression of CD11b ) ; all cytokines further increased FcRI and FcRII expression in the presence of PMA ; HLA expression was also increased in the presence of PMA, IFN alpha and IFN gamma ; dexamethasone reduced the levels of FcRI and II in cells stimulated with PMA with or without cytokines
Herrmann et al., Eur J Immunol 1992 : This suggests that high affinity Fc-Fc gamma RI interactions are required for induction of expression of GM-CSF by monocytes
Quan et al., Neurosci Lett 2009 : GM-CSF increased both FcgammaRI and FcgammaRIIb mRNA expression
Gericke et al., J Leukoc Biol 1995 : The induction of Fc gamma RI by rhG-CSF has previously been reported to result from effects on immature granulocyte progenitors
Seitz et al., Arthritis Rheum 1998 : Fluorescence activated cell sorter analysis was used to measure the changes in antigen expression ( CD11b/c, CD16, CD64 , CD14, CD68, and CD95 ) in response to MTX, 1,25-OH-cholecalciferol ( 1,25-OH-CCF ), and granulocyte-macrophage colony stimulating factor in the human monoblastic leukemia cell line U937, bone marrow mononuclear cells ( BMMC ), and peripheral blood mononuclear cells ( PBMC )