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HLA-DRB1 — IRF6

Text-mined interactions from Literome

Itoh-Lindstrom et al., J Immunol 1999 (IgG Deficiency) : Induction of MHC class II expression by IL-4 or LPS was absent on B cells, and Mac-1+ cells showed no detectable induction of MHC class II by either IL-4, LPS , or IFN-gamma
Seixas et al., Eur J Immunol 2001 : In contrast to some strains of the human parasite, P. falciparum, P.c. chabaudi ( AS ) did not inhibit the up-regulation of MHC class II , CD86 or CD40 induced by LPS
Sun et al., Vaccine 2003 : While LPS could induce high-level expression of MHC Class II and co-stimulator molecules on DCs, it did not enhance antigen presentation to co-stimulation independent DO11.GFP T hybridoma cells
Sommandas et al., J Autoimmun 2005 (Diabetes Mellitus, Type 1...) : LPS-maturation did not restore this low MHC class II expression
Kalupahana et al., Immunology 2005 : Live and heat killed salmonellae as well as lipopolysaccharide (LPS) inhibited the up-regulation of MHC class II expression induced by IFN-gamma on macrophages but not on DCs
Yao et al., J Immunol 2006 (MAP Kinase Signaling System) : In macrophages, LPS inhibited IFN-gamma-inducible CIITA and MHC class II expression without affecting expression of IFN regulatory factor-1 and MHC class I. Blocking ERK and p38 by MAPK inhibitors not only rescued LPS mediated inhibition, but also augmented IFN-gamma induction of CIITA
Zhou et al., J Immunol 2007 : Iloprost and cicaprost also suppressed LPS induced expression of CD86, CD40, and MHC class II molecules by BMDCs and inhibited the ability of BMDCs to stimulate Ag-specific CD4 T cell proliferation and production of IL-5 and IL-13
Sá-Nunes et al., J Immunol 2007 : Moreover, we have found that I. scapularis saliva ( dilution 1/200 ; approximately 10 nM PGE ( 2 ) ) marginally inhibited LPS induced CD40, but not CD80, CD86, or MHC class II expression
Politis et al., Can J Vet Res 1991 : The effect of Escherichia coli lipopolysaccharide (LPS) on the expression of major histocompatibility complex ( MHC) class II molecules by bovine mammary macrophages was examined ... Our data show that LPS had no significant effect on the expression of MHC class II molecules on the surface of mammary macrophages
Kambayashi et al., J Immunol 2009 : In this report, we demonstrate that in vitro stimulation of mouse MCs with LPS and IFN-gamma induces the expression of MHC class II and costimulatory molecules
Vilekar et al., Int Immunol 2012 : EF24 reduced the expression of LPS induced MHC class II , CD80 and CD86 molecules
Dangi et al., J Immunol 2012 : LPS increased the expression of MHC class II , CD80, and CD86 and stimulated the production of IL-1a, IL-1ß, IL-6, IL-10 and TNF-a by HSCs. Interestingly, production of IL-1a, IL-1ß, IL-6, and TNF-a was strongly inhibited, but that of IL-10 enhanced in LPS pretreated HSC/Treg cocultures
Sung et al., Biochem Biophys Res Commun 2013 : Lipopolysaccharide (LPS) induced expression of cell surface molecules ( CD80, CD86, and MHC class I/II ) and production of pro-inflammatory cytokines ( tumor necrosis factor-a, IL-1ß, IL-6, and IL-12p70 ) were inhibited by Pro B2, and this action was prevented by IRAK-M silencing
Sicher et al., J Immunol 1995 : We show that LPS may either inhibit or augment IFN-gamma induced MHC class II on macrophages depending on the sequence of the IFN-gamma and LPS signals ... LPS inhibited MHC class II when added simultaneously with IFN-gamma, but augmented class II expression when added after IFN-gamma ... However, even when NO production was inhibited, LPS given simultaneously with IFN-gamma did not augment MHC class II expression
Hart et al., J Leukoc Biol 1995 : In response to IL-10, decreases in LPS induced TNF-alpha and IL-1 beta production and reduction in the expression of major histocompatibility complex ( MHC) class II antigens were examined
Uchida et al., Int Arch Allergy Immunol 1995 : The purified lipid did not have any effect on the LPS mediated induction of MHC class II molecules on the B-cell surface
Frandji et al., J Immunol 1993 : Expression of MHC class II molecules was up-regulated upon stimulation with LPS but not with IFN-gamma and was down-regulated after exposure of mast cells to IL-3 treatment
Weber et al., J Immunol 1993 : On the other hand, the protein had little effect on LPS mediated suppression of MHC class II expression on the surface of macrophages induced with IFN-gamma
Buttini et al., Neurochem Int 1996 : Furthermore, LPS induced enhanced expression of MHC class II by microglial cells
Hart et al., Immunology 1996 : In this study, activation of human monocytes by lipopolysaccharide (LPS) prevented concomitant IL-4 stimulation of MHC class II expression ... MHC class II expression by monocytes cultured for 7 days with macrophage colony stimulating factor was regulated by IL-4 and LPS in a manner very similar to that detected for freshly isolated monocytes ... Furthermore, IFN-gamma increased MHC class II expression was suppressed by LPS , suggesting that the regulation was at the level of MHC class II expression per se
Jakob et al., J Immunol 1998 : Flow cytometry of FSDDC-A revealed that IL-1, TNF-alpha, and LPS induced increased expression of MHC class II , CD40, and CD86 and decreased E-cadherin expression that was temporally related to dissociation of aggregates
Hailer et al., Brain Pathol 1998 : Moreover, treatment of macrophages with astrocyte culture supernatant effectively down-regulated the LPS induced expression of adhesion- and MHC-class-II-molecules