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ACSL1 — IRF6

Text-mined interactions from Literome

Rubinow et al., J Biol Chem 2013 : LPS , Gram negative bacteria, IFN-?, and TNFa all induce ACSL1 expression in macrophages, whereas PPAR agonists do not ... LPS induced ACSL1 expression is dependent on Toll-like receptor 4 (TLR4) and its adaptor protein TRIF ( Toll-like receptor adaptor molecule 1 ) but does not require the MyD88 ( myeloid differentiation primary response gene 88 ) arm of TLR4 signaling ; nor does it require STAT1 ( signal transducer and activator of transcription 1 ) for maximal induction
Memon et al., Am J Physiol 1998 (Anorexia) : The inhibitory effects of LPS on ACS1 mRNA levels in liver and adipose tissue were observed as early as 2-4 h after administration, became maximal by 4-8 h, and were sustained for > /=24 h. Very low doses of LPS ( 0.1-1 microg/100 g body wt ) were needed to reduce ACS1 mRNA levels in liver and adipose tissue ... TNF and IL-1 mimicked the effect of LPS on ACS1 mRNA levels in liver and adipose tissue