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BDNF — JUN

Text-mined interactions from Literome

Ruiz-León et al., J Neurochem 2001 (Neuroblastoma) : These results suggest that activation of the APP promoter by BDNF is largely independent of PKC and AP-1
Yamagishi et al., Brain Res Mol Brain Res 2003 (Potassium Deficiency) : Comparison of inhibitory effects of brain derived neurotrophic factor and insulin-like growth factor on low potassium induced apoptosis and activation of p38 MAPK and c-Jun in cultured cerebellar granule neurons ... BDNF and IGF-1 suppressed the activation of p38 and c-Jun , but not of c-Jun N-terminal kinase (JNK), caused by lowering the potassium concentration
Wu et al., Neurobiol Dis 2012 : We found that BDNF dependent c-Jun expression and nuclear translocation required prior phosphorylation of extracellular signal regulated kinase ( ERK ) 1/2, but not Akt
Gaiddon et al., J Neurochem 1996 (Second Messenger Systems) : Here we show that BDNF stimulates AP1 binding activity in primary cerebellar neurons ... In summary, we show that BDNF stimulates AP1- and CRE dependent transcription through a mechanism that is distinct from the cAMP- and Ca ( 2+ ) -dependent pathways in CNS neurons
Sano et al., Biochem Biophys Res Commun 1996 : These results indicate that nascent AP-1 is not required for the activation of BDNF gene, leading to the notion that the BDNF gene can be activated by Ca2+ signals as an immediate early gene
Courtney et al., J Neurosci 1997 : The cells express TrkB protein and show elevated c-Fos and c-Jun levels in response to BDNF
Hsieh et al., Exp Neurol 1998 (Body Weight...) : BDNF restores the expression of Jun and Fos inducible transcription factors in the rat brain following repetitive electroconvulsive seizures