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CDH1 — TGFB2

Text-mined interactions from Literome

Chimal-Monroy et al., Int J Dev Biol 1999 : Results showed that TGF-beta1, TGF-beta2 , TGF-beta3, and TGF-beta5 differentially enhanced the expression of N-cadherin , N-CAM, fibronectin and tenascin in precartilage condensations, suggesting that TGF-beta isoforms play an important role in the establishment of cell-cell and cell-extracellular matrix interactions during precartilage condensations
Wang et al., J Cell Physiol 2001 (Colonic Neoplasms) : Requirement of protein kinase Calpha, extracellular matrix remodeling, and cell-matrix interaction for transforming growth factorbeta regulated expression of E-cadherin and catenins
Buijs et al., Am J Pathol 2007 (Bone Neoplasms...) : Exogenous addition of BMP7 to human prostate cancer cells dose-dependently inhibited transforming growth factor beta induced activation of nuclear Smad3/4 complexes via ALK5 and induced E-cadherin expression
Maretzky et al., J Invest Dermatol 2008 (Blister...) : Proinflammatory cytokines, transforming growth factor-beta , and lipopolysaccharide led to increased release of soluble E-cadherin by activating mitogen activated protein kinase signaling in cultured keratinocytes
Do et al., Mol Cancer Res 2008 (Neoplasm Metastasis...) : When cancer cells were cultured on plastic, TGF-beta1, TGF-beta2 , and TGF-beta3 induced pro-matrix metalloproteinase ( MMP ) secretion, loss of cell-cell junctions, down-regulation of E-cadherin , up-regulation of N-cadherin, and acquisition of a fibroblastoid phenotype, consistent with an epithelial-to-mesenchymal transition ... When cancer cells were cultured on plastic, TGF-beta1, TGF-beta2 , and TGF-beta3 induced pro-matrix metalloproteinase ( MMP ) secretion, loss of cell-cell junctions, down-regulation of E-cadherin , up-regulation of N-cadherin, and acquisition of a fibroblastoid phenotype, consistent with an epithelial-to-mesenchymal transition