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PRKCB — SLC2A4

Text-mined interactions from Literome

Standaert et al., Endocrinology 1999 : On the other hand, increases in 2-DOG uptake may have been partly caused by the loss of PKCbeta1, rather than PKCbeta2, as transient expression of PKCbeta1 selectively inhibited insulin stimulated translocation of epitope tagged GLUT4 in adipocytes prepared from PKCbeta knockout mice
Bandyopadhyay et al., Biochem J 1999 : In contrast with KI forms of atypical PKCs, KI forms of PKC-alpha, PKC-beta2 , PKC-delta and PKC-epsilon had little or no effect on insulin stimulated HAA-GLUT4 translocation ... Concerning the question of sufficiency, overexpression of WT-PKC-zeta enhanced insulin effects on HAA-GLUT4 translocation, whereas WT forms of PKC-alpha, PKC-beta2 , PKC-delta and PKC-epsilon did not affect GLUT4 translocation ; furthermore, Constit PKC-zeta evoked increases in HAA-GLUT4 translocation approaching those of insulin, but Constit forms of PKC-alpha and PKC-beta2 were without effect