Gene interactions and pathways from curated databases and text-mining

◀ Back to IRF6

IRF6 — TNPO1

Text-mined interactions from Literome

Mourgeon et al., Am J Physiol Lung Cell Mol Physiol 2000 : After 4 h, stretch enhanced LPS induced macrophage inflammatory protein (MIP)-2 production in a force- and frequency dependent manner
Zhang et al., J Immunol 2001 : G-CSF had no direct effect on LPS induced MIP-2 and CINC mRNA expression by alveolar macrophages
Isowa et al., Am J Physiol Lung Cell Mol Physiol 2001 : To determine whether the polymerization status of microfilaments affects LPS induced MIP-2 production, we treated rat alveolar epithelial cells with cytochalasin D ( CytoD ), a microfilament disrupting agent, before and during LPS stimulation
Kim et al., Mol Immunol 2003 : In summary, our data suggest that both NF-kappaB and c-Jun contribute to LPS induced mouse MIP-2 gene expression in RAW 264.7 cells
Miyauchi et al., Histochem Cell Biol 2004 (Periodontitis) : These findings indicated that JE cells produced MIP-2 and CINC-2 in response to LPS stimulation and suggested that MIP-2 and CINC-2 may be responsible for PMN migration toward the periodontal pathogen and may play an important role in the initiation of inflammation and subsequent periodontal tissue destruction
Husted et al., J Surg Res 2005 : LPS induced significant increases in TNFalpha and MIP-2 production
Inoue et al., FASEB J 2006 (Inflammation...) : In both genotypes of mice, LPS enhanced protein expression of interleukin (IL)-1beta, IL-6, granulocyte/macrophage-colony stimulating factor, macrophage inflammatory protein (MIP)-1alpha, MIP-2, macrophage chemoattractant protein-1, and keratinocyte chemoattractant in the lung, kidney, and liver and circulatory levels of IL-1beta, IL-6, MIP-2 , and KC
Yeh et al., Int Immunopharmacol 2006 (Disease Models, Animal...) : The results indicated that XBS down-regulated the LPS induced expression of TNF-alpha, IL-1beta, IL-6, KC, MIP-2 , and MCP-1
Kim et al., Immunol Cell Biol 2007 : A specific PPARgamma antagonist ( GW9662 ) had no effect on the inhibitory action of 15d-PGJ ( 2 ) and PGA1 in LPS induced chemokine mRNA expression and on the synergistic action of 15d-PGJ ( 2 ) in LPS induced MIP-2 ( CXCL2 ) expression ... The synergistic action of 15d-PGJ ( 2 ) on LPS induced MIP-2 ( CXCL2 ) mRNA expression was dependent on the activation of nuclear factor-kappaB (NF-kappaB), and 15d-PGJ ( 2 ) increased the phosphorylation of p38 and stress activated protein kinase/c-Jun N-terminal kinase ( SAPK/JNK ) in cells stimulated with LPS
Hauber et al., BMC pulmonary medicine 2010 : Both LPS and PAM3 significantly increased MIP-2 and TNFalpha mRNA expression compared to sham challenge ( P < 0.05 )
Eguchi et al., Clin Exp Immunol 2011 (Disease Models, Animal...) : We also show that middle ear inflammatory reactions, including infiltration of inflammatory cells and expression of MIP-2 , IL-1ß and IL-6 induced by LPS , are reduced significantly in DP? ( / ) ? mice and DP? ( / ) ? CRTH2? ( / ) ? mice
Reale et al., BMC neuroscience 2011 : In the simultaneous quantification we found significantly higher levels of constitutively and LPS induced MCP-1, MIP-1a , IL-8 and IL-18, and lower RANTES and IFN? levels released by PBMC of SC patients compared with HC
Deshpande et al., Am J Reprod Immunol 1997 : Our results show that 17 beta-estradiol decreases LPS induced IL-1 alpha, IL-6, and TNF-alpha production but not IL-10, IL-12, and macrophage inflammatory protein (MIP) production by splenic macrophages