◀ Back to FOS
FOS — TBCA
Text-mined interactions from Literome
Wei et al., Pain 1999
(Hyperalgesia...) :
Furthermore, if the NGC lesion was extended to involve the NRM, the behavioral hyperalgesia and
CFA induced
Fos expression were similar to that in vehicle injected rats
Sugiyo et al., Exp Brain Res 2001
(Hyperalgesia) :
Pretreatment with the c-fos antisense ODN significantly decreased the
CFA induced expression of
c-Fos protein dose-dependently in ipsilateral laminae I/II ( LI/II ) of the dorsal horn ( mean +/- SEM per section : 10 nM ODN, 43.9+/-1.3 ; 25 nM ODN, 19.4+/-4.1 ) compared with pretreatment with the mismatch ODN ( 63.6+/-2.9 ; 60.6+/-4.0 ) or saline ( 56.6+/-5.5 ) ... These results indicate that the expression of
CFA induced
c-Fos in the dorsal horn might facilitate thermal nociception
Loyd et al., J Comp Neurol 2006
(Inflammation...) :
Systemic administration of morphine significantly suppressed
CFA induced
Fos in the PAG in males only
Xiao et al., Brain Res 2008
(Hyperalgesia...) :
In addition, the antisense knockdown of ERK5 reduced
CFA induced phosphorylation of cAMP response-element binding protein ( CREB ), a downstream substrate of the ERK5 pathway, and expression of
Fos , a marker for neuronal activation in the central nervous system
Yang et al., Anat Rec (Hoboken) 2010
(Hyperalgesia) :
In addition, TENS application suppressed the
CFA induced ERK2 activation and
c-Fos protein expression
Li et al., Am J Chin Med 2010
(Arthritis, Experimental...) :
In addition,
CFA induced
Fos expression in the superficial layer of the lumbar spinal cord was significantly suppressed by the BV and melittin treatments, compared to the saline treatment
Marchand et al., Molecular pain 2011
(Inflammation...) :
Finally, inhibition of PKC?/PKM? activity decreased the expression of
Fos in
response to formalin and
CFA in both superficial and deep laminae of the dorsal horn
Ren et al., Proc Natl Acad Sci U S A 1997
(Hyperalgesia...) :
Parallel findings were obtained for
CFA induced
Fos-like immunoreactivity (Fos-LI) , a marker of neuronal activation