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IL18 — IL3

Text-mined interactions from Literome

Zirngibl et al., Mol Cell Biol 2002 : Bone marrow derived Fps/Fes-null macrophages showed no defects in granulocyte-macrophage colony stimulating factor-, interleukin 6 (IL-6)-, or IL-3 induced activation of signal transducer and activator of transcription 3 ( Stat3 ) and Stat5A or LPS induced degradation of I kappa B or activation of p38, Jnk, Erk, or Akt
Takahashi et al., Antimicrob Agents Chemother 2005 : In addition, ciprofloxacin suppressed the interleukin-18 induced production of tumor necrosis factor alpha, gamma interferon, and interleukin-12 in peripheral blood mononuclear cells by inhibiting the expression of intercellular adhesion molecule 1, B7.1, B7.2, and CD40 on monocytes, and this effect could be reversed by the addition of indomethacin or NS398
Matsuzaki et al., Cancer Sci 2006 : We have demonstrated previously that the Th1-cytokine conditioned bone marrow derived dendritic cell ( BMDC ) subset BMDC1 ( generated in the presence of granulocyte-macrophage colony stimulating factor [GM-CSF ] + interleukin [ IL]-3 + interferon [ IFN]-gamma+ IL-12 ) induces a much stronger type 1 immune response than BMDC0 ( GM-CSF + IL-3 )
Terada et al., Proc Natl Acad Sci U S A 2006 (Dermatitis, Atopic...) : Blockade of IL-18 prevented AD development, whereas blockade of IL-3 partially prevented AD development, suggesting a contribution of IL-18 dependent IL-3 production to AD with cutaneous mastocytosis
Cohen et al., Immunol Lett 1991 : We have studied the production of interleukin-1 (IL-1), interleukin-6 (IL-6) and tumor necrosis factor (TNF) by mouse peritoneal macrophages triggered by lipopolysaccharide (LPS) in the presence or absence of IL-3
Cavaillon et al., Cell Immunol 1990 : Induction of interleukin-3 by interleukin-1 in the absence of other exogenous stimuli