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PLG — QRICH2

Text-mined interactions from Literome

Goldberg et al., J Biol Chem 2002 : We previously demonstrated in glomerular mesangial and endothelial cells that both high glucose concentrations and glucosamine activated the plasminogen activator inhibitor-1 ( PAI-1 ) gene promoter through the transcription factor, Sp1 ; and that the glutamine : fructose-6-phosphate amidotransferase inhibitor, 6-diazo-5-oxonorleucine, inhibited the effect of high glucose, but not that of glucosamine
Thompson et al., Mol Cell Endocrinol 1979 (Cell Transformation, Viral...) : HTC cell variants chosen for their lack of tyrosine aminotransferase ( EC 2.6.1.5 ) ( TAT ) induction by glucocorticoids were tested for interrelated effects on other glucocorticoid responses : TAT induction by dibutyryl cyclic AMP ( dBcAMP ) +/- dexamethasone, glutamine synthetase ( GS ) induction, cyclic nucleotide phosphodieterase ( PDE ) suppression, inhibition of alpha-aminoisobutyric acid ( AIB ) uptake, inhibition of plasminogen activator (PA) , and induction of mouse mammary tumor virus ( MTV )