- Home
- Genomes
- Genome Browser
- Tools
- Mirrors
- Downloads
- My Data
- Projects
- Help
- About Us
Notch → LPS: " The expression of Notch target genes including Hes1 and Hes5 was also induced in macrophages by LPS , suggesting that TLR4 signaling enhances the activation of Notch pathway "
IL-10 ⊣ Notch1: " Importantly, overexpression of constituted active form of Notch1 ( NICD1 ) and Notch2 ( NICD2 ) suppressed production of TLR4 triggered proinflammatory cytokines such as TNF-a and IL-6 but promoted production of antiinflammatory cytokine IL-10 , which is dependent on the PEST domain of NICD "
IL-10 ⊣ Notch2: " Importantly, overexpression of constituted active form of Notch1 ( NICD1 ) and Notch2 ( NICD2 ) suppressed production of TLR4 triggered proinflammatory cytokines such as TNF-a and IL-6 but promoted production of antiinflammatory cytokine IL-10 , which is dependent on the PEST domain of NICD "
TNF-a → Notch1: " Importantly, overexpression of constituted active form of Notch1 ( NICD1 ) and Notch2 ( NICD2 ) suppressed production of TLR4 triggered proinflammatory cytokines such as TNF-a and IL-6 but promoted production of antiinflammatory cytokine IL-10, which is dependent on the PEST domain of NICD "
TNF-a → Notch2: " Importantly, overexpression of constituted active form of Notch1 ( NICD1 ) and Notch2 ( NICD2 ) suppressed production of TLR4 triggered proinflammatory cytokines such as TNF-a and IL-6 but promoted production of antiinflammatory cytokine IL-10, which is dependent on the PEST domain of NICD "
IL-6 → Notch1: " Importantly, overexpression of constituted active form of Notch1 ( NICD1 ) and Notch2 ( NICD2 ) suppressed production of TLR4 triggered proinflammatory cytokines such as TNF-a and IL-6 but promoted production of antiinflammatory cytokine IL-10, which is dependent on the PEST domain of NICD "
IL-6 → Notch2: " Importantly, overexpression of constituted active form of Notch1 ( NICD1 ) and Notch2 ( NICD2 ) suppressed production of TLR4 triggered proinflammatory cytokines such as TNF-a and IL-6 but promoted production of antiinflammatory cytokine IL-10, which is dependent on the PEST domain of NICD "