Gene interactions and pathways from curated databases and text-mining

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MAPK3 — NTRK3

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

Text-mined interactions from Literome

Tognon et al., Cancer Res 2001 (Cell Transformation, Neoplastic...) : The chimeric protein tyrosine kinase ETV6-NTRK3 requires both Ras-Erk1/2 and PI3-kinase-Akt signaling for fibroblast transformation
Jin et al., Cancer Res 2007 (Cell Transformation, Neoplastic) : Here, we define some aspects of the molecular mechanisms regulating ETV6-NTRK dependent Ras-Erk1/2 and PI3K-Akt activation
Jin et al., J Biol Chem 2008 (Cell Transformation, Neoplastic) : Here we define some aspects of the molecular mechanisms regulating TrkC dependent Ras-Erk1/2 and PI3K/Akt activation
Weinkauf et al., Infect Immun 2009 (Chagas Disease) : PDNF promotes TrkC dependent mitogen activated protein kinase signaling, neurite outgrowth, and survival of genetically engineered PC12 neuronal cells and glial Schwann cells in a TrkC dependent manner
Ivanov et al., Oncogene 2013 (Carcinoma, Adenoid Cystic...) : NT-3 stimulation of U2OS cells with ectopic TrkC expression triggered TrkC phosphorylation and resulted in Ras, Erk 1/2 and Akt activation, as well as VEGFR1 phosphorylation
Guiton et al., J Biol Chem 1995 : In cells expressing full-length TrkC, neurotrophin-3 promoted a sustained activation of mitogen activated protein kinase ; TrkC containing kinase inserts only mediated transient activation of mitogen activated protein kinase
Gunn-Moore et al., Biochem J 1997 : Experiments with the use of cell-permeant inhibitors argue against a major role for PLC gamma and PtdIns 3-kinase in the activation of MAPK by TrkC