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IL17A — IL22

Text-mined interactions from Literome

Sonnenberg et al., J Exp Med 2010 (Pneumonia) : However, despite increased bleomycin induced IL-22 production, Il17a-/- mice were protected from airway inflammation, suggesting that IL-17A may regulate the expression and/or proinflammatory properties of IL-22 ... Consistent with this, IL-17A inhibited IL-22 production by Th17 cells, and exogenous administration of IL-22 could only promote airway inflammation in vivo by acting in synergy with IL-17A
Cella et al., Proc Natl Acad Sci U S A 2010 : IL-1beta promoted constitutive IL-22 secretion rather than acute IL-22 production in response to IL-23 and induced IL-17 in some cells
Freches et al., Clinical and vaccine immunology : CVI 2011 (Tuberculosis) : Increased pulmonary tumor necrosis factor alpha, interleukin-6 (IL-6), and IL-17A responses compensate for decreased gamma interferon production in anti-IL-12 autovaccine treated, Mycobacterium bovis BCG vaccinated mice
Baba et al., Hum Immunol 2012 : Human monocytes exposed to VAF347 differentiated into dendritic cells capable of instructing a naïve CD4 ( + ) T cell differentiation program that promoted IL-22 secretion and concomitantly inhibited IL-17 production
Nishimori et al., Infect Immun 2012 (Colitis...) : Microbial amyloids induce interleukin 17A (IL-17A) and IL-22 responses via Toll-like receptor 2 activation in the intestinal mucosa
van Hamburg et al., Ann Rheum Dis 2013 (Arthritis, Experimental...) : In T cell-RASF co-cultures, IL-22 in the presence of IL-17A had limited effects on IL-6, IL-8, matrix metalloproteinase-1 (MMP-1) and MMP-3 production
Sarkar et al., Arthritis Rheum 2013 (Arthritis, Experimental...) : IL-22 augmented IL-10, IL-17 , and IL-6 in lymphoid tissues in vitro
Kong et al., Mol Med Report 2013 : We have previously demonstrated that IL-22 has critical anti-inflammatory and antiviral roles in mice with coxsackievirus B3 ( CVB3 ) -induced acute viral myocarditis ( AVMC ) in the presence of IL-17A ... Therefore, the present study, in continuation of our previous investigations, aimed to determine whether IL-22 plays a distinctly different role in the absence of IL-17A in AVMC by using IL-17A-deficient mice