Gene interactions and pathways from curated databases and text-mining

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GYS1 — WNT11

Text-mined interactions from Literome

Iseki et al., Cancer Sci 2010 : Interestingly, luciferase reporter analysis and quantitative real-time RT-PCR demonstrated that the ALEX1 promoter was up-regulated in a CRE dependent manner by continuous activation of Wnt/beta-catenin signaling induced by a glycogen synthase kinase-3 inhibitor and overexpression of beta-catenin
Gambardella et al., J Bone Miner Res 2011 : Activation of canonical Wnt signaling by inhibition of glycogen synthase kinase 3 ( GSK-3 ) has been shown to enhance bone mass, possibly by involving a number of mechanisms ranging from amplification of the mesenchymal stem cell pool to the commitment and differentiation of osteoblasts
Dobrowolski et al., Cell reports 2012 : Sustained canonical Wnt signaling requires the inhibition of glycogen synthase kinase 3 ( GSK3 ) activity by sequestration of GSK3 inside multivesicular endosomes ( MVEs )