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AKT1 — F2RL3

Text-mined interactions from Literome

Reséndiz et al., Journal of thrombosis and haemostasis : JTH 2007 : Stimulation of PAR1 or PAR4 resulted in rapid Akt phosphorylation, independently of secreted ADP and phosphatidylinositol-3-kinase (PI3K) activation
Li et al., J Biol Chem 2011 (Thrombosis) : The function of arrestin-2 in platelets is agonist-specific, as PAR4 dependent Akt phosphorylation and fibrinogen binding were reduced in arrestin-2 knock-out platelets compared with WT controls, but ADP stimulated signaling to Akt and fibrinogen binding were unaffected