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ETS1 — NOS1

Text-mined interactions from Literome

Rife et al., Brain Res Mol Brain Res 2000 : Thus, it is possible that activation of Ets and/or AP1 transcription factors by the Ras-Raf-MAP kinase cascade contributes to the NGF mediated induction of nNOS
Takahashi et al., Mol Cell Biochem 2001 : Ets-1 is involved in transcriptional regulation of the chick inducible nitric oxide synthase gene in embryonic ventricular myocytes
Di Nunzio et al., Am J Physiol Regul Integr Comp Physiol 2002 : In addition, both ETs increased nitric oxide synthase activity
Jaureguiberry et al., Peptides 2004 : Results showed that ETs stimulated NOS activity was inhibited by a selective ETB antagonist ( BQ-788 ), but not by a selective ETA antagonist ( BQ-610 ) ... Both Nomega-nitro-L-arginine methyl ester ( NOS inhibitor ), and 7-nitroindazole ( neuronal NOS inhibitor ) diminished ETs stimulated NOS activity ... The stimulatory effect of ETs on NOS activity was inhibited in the presence of PLC, PKC, PKA and CaMK-II inhibitors ( U-73122, GF-109203X, H-89 and KN-62, respectively ), and the IP3 receptor selective antagonist, 2-APB
Anita et al., Cell Mol Neurobiol 2006 : 1. We further characterized the effect of endothelins ( ETs ) on receptor mediated phosphoinositide ( PI ) turnover, nitric oxide synthase (NOS) activation, and cGMP formation in whole rat adrenal medulla ... 1. We further characterized the effect of endothelins ( ETs ) on receptor mediated phosphoinositide ( PI ) turnover, nitric oxide synthase (NOS) activation, and cGMP formation in whole rat adrenal medulla