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EPHA3 — STAT3
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Tsai et al., Mol Cell Biol 2000
(Cell Transformation, Neoplastic) :
To investigate whether
Etk could
mediate v-Src induced activation of
STAT3 and cell transformation, we overexpressed a dominant negative mutant of Etk in an immortalized, untransformed rat liver epithelial cell line, WB, which contains endogenous Etk
Guo et al., PloS one 2011
(Neoplasm Invasiveness...) :
We found that down-regulation of
ETK in bladder cancer cells
attenuated STAT3 and AKT activity whereas exogenous overexpression of ETK had opposite effects, suggesting that deregulation of ETK may attribute to the elevated activity of STAT3 and AKT frequently detected in bladder cancer