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EDN2 — NOS1

Text-mined interactions from Literome

Barton et al., J Am Soc Nephrol 2000 (Glomerulosclerosis, Focal Segmental...) : Dysfunctional renal nitric oxide synthase as a determinant of salt-sensitive hypertension : mechanisms of renal artery endothelial dysfunction and role of endothelin for vascular hypertrophy and Glomerulosclerosis
Zhan et al., Sheng li ke xue jin zhan [Progress in physiology] 2000 (Cardiomyopathy, Hypertrophic) : Angiotensin II ( AII ), endothelin-1 (ET-1) and norepinephrine ( NE ) can inhibit NOS activity and NO production, and induce hypertrophic response in cultured neonatal rat cardiomyocytes ; these effects of AII, ET-1 and NE are mediated respectively by AII receptor, ETA receptor and alpha 1-adrenergic receptor ; these effects of AII and ET-1 are mediated by PTX-sensitive G protein, while the effects of NE are mediated by PTX-insensitive G protein
Verma et al., J Surg Res 2004 (Anoxia) : In some experiments, the effects of endothelin ( ET ) receptor blockade ( with bosentan ) and nitric oxide synthase (NOS) inhibition ( with L-NAME ) on pravastatin mediated cardioprotection were evaluated
Ohlstein et al., J Neurosurg 1992 : An l-arginine analog inhibitor of nitric oxide synthase , L-NG-monomethyl arginine ( L-NMMA, 200 microM ), did not significantly affect basal immunoreactive endothelin levels
Novella et al., Eur J Cardiothorac Surg 2007 (Cardiovascular Diseases) : No change in the AVP response was produced by endothelium removal, exposure to the NO precursor ( 3 x 10 ( -4 ) M L-arginine ), inhibition of nitric oxide ( NO ) synthase ( 3 x 10 ( -5 ) M L-NAME ) or soluble guanylate cyclase ( 3 x 10 ( -6 ) M 1H- [ 1,2,4 ] oxadiazol [ 4,3,-alpha ] quinoxalin-1-one ( ODQ ) ), removal of the superoxide anion ( 100 U/ml superoxide dismutase ( SOD ) plus 1200 U/ml catalase ) or hydroxyl radical ( 10 ( -4 ) M deferoxamine ), or specific alpha1 - ( 10 ( -6 ) M prazosin ) or endothelin ( 10 ( -5 ) M bosentan ) receptor antagonism
Franchi et al., Proc Natl Acad Sci U S A 1994 : Cholinergic stimulation of vascular endothelin activates NO synthase (NOS) , leading to generation of NO from arginine
Lin et al., Eur J Endocrinol 1998 : Role of nitric oxide synthase in release of endothelin from cultured human endometrial cells