Day et al., J Cell Biol 2010
:
Efficient association of Rad18 with Pol? is dependent on DDK and is necessary for redistribution of Pol? to sites of replication fork stalling
Vaziri et al., Cell cycle (Georgetown, Tex.) 2010
:
DDK mediated Rad18 phosphorylation promotes Rad18-Pol? complex formation and facilitates Rad18 dependent recruitment of Pol? to stalled replication forks