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CASP7 — CXCL9

Text-mined interactions from Literome

You et al., Hepatology 2001 (Carcinoma, Hepatocellular...) : Furthermore, apoptosis could be completely blocked by Z-VAD-FMK ( a general caspase inhibitor ) or by IETD-CHO ( a caspase-8 inhibitor ), but was only partially blocked by Ac-DEVD-CMK ( a caspase-3 inhibitor ), by N-acetyl-L-cysteine (NAC) ( an antioxidant ), by N-acetyl-leucyl-leucyl-norleucinal ( ALLN ) ( a calpain inhibitor I ), or by Z-LEHD-FMK ( a caspase-9 inhibitor )
Latorre et al., Eur J Biochem 2003 : Furthermore, experiments with Ac-DEVD-CMK ( caspase-3 specific inhibitor ) have led us to conclude that caspase-3 is downstream of ceramide in activating the brain plasmalogen-selective phospholipase A2
Florio et al., J Cell Biochem 2003 (Leukemia, Lymphocytic, Chronic, B-Cell) : Furthermore, both caspase-3 activation and apoptosis triggered by IDA alone or in combination with MPA were significantly inhibited by specific caspase-3 inhibitor AC-DEVD-CMK
Chen et al., Mol Endocrinol 2013 (Diabetes Mellitus, Type 2) : Using adenoviral vectors to overexpress or knockdown Mig6, we found that caspase 3 activation during apoptosis was dependent on the level of Mig6
Iwasaki et al., Biochem Mol Biol Int 1997 (Carcinoma, Squamous Cell) : Both YVAD-CMK , an inhibitor of caspase-1 , and DEVD-CHO, an inhibitor of caspase-3, moderately inhibited the UVB induced cell death