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CSK — STAT3

Text-mined interactions from Literome

Ram et al., Science 2000 (Cell Transformation, Neoplastic...) : Furthermore, Galphao* expression increased activity of the tyrosine kinase c-Src, and the Galphao* induced activation of Stat3 was blocked by expression of Csk ( carboxyl-terminal Src kinase ), which inactivates c-Src
Hung et al., J Biol Chem 2001 (Mammary Neoplasms, Experimental) : c-Src kinase activity correspondingly increased the tyrosine 705 phosphorylation and DNA binding affinity of Stat3 ( but not Stat1, -5A, or -5B )
Hwang et al., Mol Endocrinol 2003 (Carcinoma, Papillary...) : Activation of STAT3 requires the intrinsic kinase activity of RET/PTC ; Janus tyrosine kinase and c-Src kinase are not involved in the RET/PTC mediated activation of STAT3
Park et al., J Cell Biochem 2013 : Pam3 CSK4 stimulated STAT3 phosphorylation ( S727 ), while STAT3 knock-down by siRNA reduced Pam3 CSK4 induced TF expression ... RGS2 knock-down elevated Pam3 CSK4 induced STAT3 phosphorylation, but RGS2 overexpression had the opposite effect on STAT3 phosphorylation