Gene interactions and pathways from curated databases and text-mining

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CCL11 — MAPK3

Pathways - manually collected, often from reviews:

Text-mined interactions from Literome

Cui et al., Am J Respir Cell Mol Biol 2002 : Eotaxin ( 1-100 nM ) induced tyrosine/threonine phosphorylation and activation of extracellular regulated kinase (ERK) 1/2 and p38 in NCI-H ( 292 ) cells and normal human bronchial epithelial cells
Rokudai et al., Biol Pharm Bull 2006 (Asthma...) : Eotaxin-1/CCL11 production was inhibited by a p38 mitogen activated protein kinase ( MAPK ) inhibitor, SB203580, but not by the MEK ( MAPK/ERK kinase ) inhibitors, PD98059 and U0126
Clements et al., J Immunol 2010 (Angiomyolipoma...) : In AML cells, their ligands CXCL12 CX3CL1, CCL11 , CCL24, and CCL28 caused robust phosphorylation of p42/44 MAPK and Akt
Zhou et al., J Immunol 2012 (Asthma...) : MAPK regulation of IL-4/IL-13 receptors contributes to the synergistic increase in CCL11/eotaxin-1 in response to TGF-ß1 and IL-13 in human airway fibroblasts
Markwick et al., Clin Exp Allergy 2012 (Asthma) : CCR3 ligands CCL11 and CCL24 caused rapid activation of p42/44 MAPK but not Akt