◀ Back to IGF1

CA2 — IGF1

Text-mined interactions from Literome

Pecherskaya et al., Alcohol Clin Exp Res 2002 (Alcoholism) : Here, we report that in alcohol exposed myocytes, IGF-I induced augmentation of the cardiac Ca2+ channel activity was absent ... Here, we report that in alcohol exposed myocytes, IGF-I induced augmentation of the cardiac Ca2+ channel activity was absent
Gonzalez et al., J Physiol 2003 : Insulin-like growth factor-1 prevents age related decrease in specific force and intracellular Ca2+ in single intact muscle fibres from transgenic mice ... Therefore, IGF-1 prevents age related decline in peak intracellular Ca2+ and specific force in a muscle that does not exhibit changes in fibre type composition with senescence
Fant et al., J Clin Endocrinol Metab 1992 : IGF-I , however, had no potentiating effect on Et-1 stimulated phosphoinositide turnover or increase in cytosolic Ca2+
Kojima et al., Am J Physiol 1992 : In medium containing 10 microM extracellular calcium, IGF-I did not cause any increase in [ Ca2+ ] c. Likewise, blockade of IGF induced calcium entry by either cobalt or tetramethrin abolished IGF-I action on [ Ca2+ ] c. IGF-I mediated oscillation was not affected by either ryanodine or caffeine, compounds that affect calcium induced calcium release
Jaimovich et al., Biol Res 2004 (Calcium Signaling) : We studied the effect of IGF-I , insulin and testosterone on intracellular Ca2+ in cultured muscle cells
Takasu et al., Biochem Biophys Res Commun 1991 : Insulin-like growth factor I (IGF-I) increased cytoplamic pH ( pHi ) and cytoplasmic Ca2+ [ ( Ca2+ ] i ) in cultured porcine thyroid cells
Muller et al., Biochem J 1991 : The effects of T3 plus IGF-I on Ca2 ( + ) -ATPase levels and Ca2+ uptake activity were more than additive ... The enhanced effect of T3 on Ca2 ( + ) -ATPase net synthesis and Ca2+ uptake activity in the presence of IGF-I can not therefore be explained by an increased myotube formation stimulated by the latter
Levy et al., Endocrinology 1986 : IGF-I increased ( Ca2+ + Mg2+ ) ATPase activity by 40 % ( P less than 0.05 ) at 0.1 microM free Ca2+ at high peptide concentrations ( 10 ng/ml )
Nishimoto et al., Biochem Biophys Res Commun 1987 : Without EGF treatment, IGF-II does not cause any increase in [ Ca2+ ] c
Poiraudeau et al., J Cell Biochem 1997 : 2 ) IGF-1 , IGF-2, and insulin also induced a Ca2+ mobilization from the endoplasmic reticulum : phospholipase C (PLC) inhibitors, neomycin, or U-73122 partially blocked the intracellular [ Ca2+ ] i increase induced by IGF-1 and insulin and totally inhibited the effect of IGF-2
Touyz et al., Hypertension 1997 : PDGF-BB and IGF-1 ( 1 to 10 ng/mL ) increased [Ca2+ ] i and pHi in a dose dependent manner
Torres-Aleman et al., Neuroscience 1998 : In addition, insulin-like growth factor-I increased intracellular Ca2+ levels in Purkinje cells and c-Fos in dividing glioblasts