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ATP5O — NOS1

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Holton et al., Cardiovasc Res 2010 : Endothelial nitric oxide synthase activity is inhibited by the plasma membrane calcium ATPase in human endothelial cells
Javkhedkar et al., American journal of physiology. Renal physiology 2012 : In the presence of N ( G ) -nitro-l-arginine methyl ester ( l-NAME ), a nitric oxide ( NO ) synthase ( NOS ) inhibitor, ANG II ( µM ) caused stimulation of Na-K-ATPase in proximal tubules of WKY rats while having no further stimulatory effect in SHR. ANG II ( µM ), via AT1R, increased proximal tubular NO levels in WKY rats but not in SHR
Groenendaal et al., Biol Neonate 1995 (Acidosis, Lactic...) : The results demonstrate that inhibition of the cerebral nitric oxide synthase activity in vivo in newborn piglets by intravenous administration of NNLA did not affect the cortical cell membrane Na+, K ( + ) -ATPase activity up to a dose of 75 mg/kg
Groenendaal et al., Pediatr Res 1997 (Hypoxia, Brain) : The present study tested the hypothesis that inhibition of NO synthase (NOS) reduced hypoxia induced modifications of Na+, K+-ATPase activity, lipid peroxidation, and [ 3H ] MK-801 binding to the N-methyl-D-aspartate ( NMDA ) receptor in cerebral cortical tissue of newborn piglets
Nicoletti et al., Biochimie 1998 : In the present study we evaluated the effects of NO synthase (NOS) induction on the regulation of cytochrome c oxidase ( CO ) and F0F1-ATPase subunit expression in astroglial and mixed cortical cell cultures