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AKT1 — ARSK

Text-mined interactions from Literome

Wu et al., Neurosci Lett 2006 (Brain Ischemia) : Down-regulation of PTEN by sodium orthovanadate inhibits ASK1 activation via PI3-K/Akt during cerebral ischemia in rat hippocampus
Taniike et al., Circulation 2008 (Cardiomegaly...) : Furthermore, the inhibition of ASK1 or p38 activity enhanced insulin-like growth factor 1-induced protein synthesis in rat neonatal ventricular cardiomyocytes, and the treatment with a specific inhibitor of p38 resulted in enhancement of Akt activation and suppression of protein phosphatase 2A activation
Xu et al., Neuropharmacology 2008 (Brain Ischemia...) : In particular, our study showed that co-activation of GABA A and GABA B receptors could strongly increase Akt activation and inhibit ASK1 activation by phosphorylation of serine 83 of ASK1
Xie et al., Proc Natl Acad Sci U S A 2009 (Prostatic Neoplasms) : Gain of function study showed that DAB2IP can suppress the PI3K-Akt pathway and enhance ASK1 activation leading to cell apoptosis, whereas loss of DAB2IP expression resulted in PI3K-Akt activation and ASK1-JNK inactivation leading to accelerated PCa growth in vivo
Pan et al., Chem Res Toxicol 2010 : Silencing Akt by siRNA reduced the activation of ASK1 and p38 MAPK and cell apoptosis, whereas without nickel stimulation, siRNA Akt had no effect on the activation of ASK1 and p38 MAPK
Park et al., Cell Mol Life Sci 2010 (Neuroblastoma) : This regulatory action of DSUP13 on ASK1 activity involves competition with Akt1 , a negative regulator of ASK1 , for binding to ASK1